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Dept. Head: Univ.-Prof.
Dr. Michaela Kress |
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The goal of physiology is to explain the physical and chemical factors that are responsible for the origin, development and progression of life. Basic research in physiology explores normal cell, organ, and whole body function. The major tasks of the Division of Physiology at Innsbruck Medical University are the teaching of human physiology and pathophysiology, the advancement of the knowledge base of these subjects by research of basic physiological and pathophysiological mechanisms, and the training of graduate students in bio-medical sciences. Physiologie ist die Lehre der Lebensvorgänge, die Physiologie lehrt und erforscht die normalen Organ- und Körperfunktionen. Die Aufgaben der Sektion für Physiologie der Medizinischen Universität Innsbruck umfassen die Lehre der Physiologie und Pathophysiologie des Menschens, die Weiterentwicklung des Wissensstandes durch die Erforschung grundlegender physiologischer wie auch pathophysiologischer Mechanismen, sowie die Ausbildung der Fachärzte und des wissenschaftlichen Nachwuchses auf dem Gebiet der biomedizinischen Forschung. |
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Research Groups
Johannes Fürst »» Gerhard Gstraunthaler »» Thomas Haller »» Walter Pfaller »» Harald Völkl »» Neurophysiology Bernhard Flucher »» Michaela Kress »» |
Publications
All Publications from the Division of Physiology Innsbruck (via Pubmed search)
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Highlighted Publications
Fusion-Activated Ca Entry: An "Active Zone" of Elevated Ca during the Postfusion Stage of Lamellar Body Exocytosis in Rat Type II Pneumocytes.
Miklavc P. et. al. PlosOne 2010 Jun 8;5(6):e10982.
A key role for gp130 expressed on peripheral sensory nerves in pathological pain.
Andratsch M. et. al.
J Neurosci. 2009 Oct 28;29(43):13473-83. PMID 19864560
Inhibitor {kappa}B Kinase {beta} Deficiency in Primary Nociceptive Neurons Increases TRP Channel Sensitivity.
Bockhart V et. al.
J Neurosci. 2009 Oct 14;29(41):12919-29. PMID: 19828806
Stable membrane expression of postsynaptic CaV1.2 calcium channel clusters is
independent of interactions with AKAP79/150 and PDZ proteins. Di Biase V et. al.
J Neurosci. 2008 Dec 17;28(51):13845-55. PMID: 19091974
PKC-theta selectively controls the adhesion-stimulating molecule Rap1. Letschka T et. al.
Blood. 2008 Dec 1;112(12):4617-27. PMID: 18796635
Endogenous tumor necrosis factor alpha (TNFalpha) requires TNF receptor type 2 to generate heat hyperalgesia in a mouse cancer model.
Constantin CE et. al. J Neurosci. 2008 May 7;28(19):5072-81.
Computer modeling of siRNA knockdown effects indicates an essential
role of the Ca2+ channel α2δ-1 subunit in cardiac
excitation-contraction coupling.
Tuluc P et. al.
Proc Natl Acad Sci U S A. 2007 Jun 26;104(26):11091-6. Epub 2007 Jun 11
Cannabinoids mediate analgesia largely via peripheral type 1 cannabinoid receptors in nociceptors. Agarwal N et. al.
Nat Neurosci. 2007 Jul;10(7):870-9
Membrane Targeting and Secretion of Mutant Uromodulin in Familial Juvenile Hyperuricemic Nephropathy
Jennings et. al. J Am Soc Nephrol 18: 264-273, 2007
Last updated
06/12/2010
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© 2007 Paul Jennings